NAME: Wang, Liang

POSITION TITLE: Lecturer of Medical Genetics, Shenzhen University School of Medicine

EDUCATION/TRAINING

A.   Personal Statement

I am a Lecturer of Medical Genetics and Molecular Genetics in the School of Medicine, Shenzhen University.My research interests lie in cell biology and tumorigenesis. I have been supported by one grant from the General Program of National Natural Science Foundation in China (NSFC), and threegrants from the Science and Technology Bureau of Shenzhen City grant.

B.   Positions and Honors

Positions and Employment

0 9 /20 08 – present       Lecturer, Shenzhen University, Shenzhen, China

C.   Research Support

Completed Research Support

NSFC (China) (81171921)          01/01/2012 to 12/31/2012

Liang Wang, PI                            ￥ 140,000

The role of KLF4 phosphorylation by CDK3 in cell transformation and tumorigenesis

The Science and Technology Bureau of ShenZhen City grant (JC201006010727A)       03/06/2011 to 03/05/2014

Liang Wang, PI                            ￥ 100,000

Corelation research of allergic rhinitis and caveolin-1 expression level and its methylation,

The Science and Technology Bureau of ShenZhen City grant (JCYJ20120613165853326): 06/01/2013 to 05/31/2015

Liang Wang, PI                            ￥ 100,000

The role of KLF4 in cell transformation and tumorigenesis,

The Science and Technology Bureau of ShenZhen City grant (JCYJ20150525092940973) 09/25/2015 to 09/30/2017

Liang Wang, PI                            ￥ 300,000

The role of MAL expression profile in cell apoptosis.

D.   Peer-reviewed publications (*: corresponding author)

1.    Jin Z, Wang L , Zhang Y, Cheng Y, Gao Y, Feng X, Dong M, Cao Z, Chen S, Yu H,Zhao Z, Zhang X, Liu J, Mori Y, Fan X, Meltzer SJ. MAL hypermethylation is atissue-specific event that correlates with MAL mRNA expression in esophagealcarcinoma. Sci Rep. 2013. 3;3:2838. doi: 10.1038/srep02838. 

2.    Wang L , Hu HY, Lin YL, Zhao ZX, Tan L, Yu P, Wan HJ, Jin Z, Zheng D. CDK3 expression and its clinical significance in human nasopharyngeal carcinoma. Mol Med Rep. 2014 Jun;9(6):2582-6. doi: 10.3892/mmr. 2095. Epub 2014 Mar 31. 

3.    Jin Z, Wang L , Cao Z, Cheng Y, Gao Y, Feng X, Chen S, Yu H, Wu W, Zhao Z, Dong M, Zhang X, Liu J, Fan X, Mori Y, Meltzer SJ. Temporal evolution in caveolin 1 methylation levels during human esophageal carcinogenesis. BMC Cancer. 2014 May 20;14:345. doi: 10.1186/1471-2407-14-345. 

4.    Wang L , Liu YJ, Xiao P, Shen H, Deng HY, Papasian CJ, Drees BM, Hamilton JJ, Recker RR, Deng HW. Chromosome 2q32 may harbor a QTL affecting BMD variation atdifferent skeletal sites. J Bone Miner Res. 2007 Nov;22(11):1672-8. PubMed PMID: 17680728.

5.    Jin Z, Feng X, Jian Q, Cheng Y, Gao Y, Zhang X, Wang L , Zhang Y, Huang W, Fan X, Chen S, Yu H, Zhao Z, Dong M, Liu J, Mori Y, Meltzer SJ. Aberrant methylation of the Ras-related associated with diabetes gene in human primary esophagealcancer. Anticancer Res. 2013. 33(11):5199-203.

6.    Jin Z, Zhao Z, Cheng Y, Dong M, Zhang X, Wang L , Fan X, Feng X, Mori Y,Meltzer SJ. Endoglin promoter hypermethylation identifies a field defect in humanprimary esophageal cancer. Cancer. 2013. 15;119(20):3604-9.